X-Message-Number: 13412 From: Date: Wed, 22 Mar 2000 00:43:22 -0800 Subject: Re: CryoNet #13408 - #13410 The confernece on 01 April will be at Stanford University, right? John Perry On Tue, 21 Mar 2000 05:00:02 -0500 (EST) CryoNet <> writes: > CryoNet - Tue 21 Mar 2000 > > #13408: ANNOUNCE: Kurzweil/Moravec Symposium: "Will Spiritual > Robots Replace Humanity by 2100?" [John Clark] > #13409: Charles, you just gotta believe! [john grigg] > #13410: peg - a non-toxic treatment for colon cancer [Doug > Skrecky] > > ---------------------------------------------------------------------- > > Message #13408 > From: "John Clark" <> > Subject: ANNOUNCE: Kurzweil/Moravec Symposium: "Will Spiritual > Robots Replace Humanity by 2100?" > Date: Mon, 20 Mar 2000 13:19:12 -0500 > > "Will Spiritual Robots Replace Humanity by 2100?" > ---------------- > > Douglas Hofstadter presents... > Kurzweil/Moravec Symposium > FREE and open to the public > April 1, 2000, 1pm - 5:30. TCSEQ room 200. > (Parking in "A" lots OK on Saturdays) > > Primary speakers: > * Ray Kurzweil (inventor of reading machine for the blind, > electronic keyboards, etc., and author of "The Age of Spiritual > Machines") > * Hans Moravec (a pioneer of mobile robot research, and author of > "Robot: Mere Machine to Transcendent Mind") > * Bill Joy (co-founder of, and chief scientist at, SUN Microsystems) > > > Panel members: > * Ralph Merkle (well-known computer scientist and one of today's top > figures in the explosive field of nanotechnology) > * Kevin Kelly (editor at "Wired" magazine and author of "Out of > Control", a study of bio-technological hybrids) > * John Holland (inventor of genetic algorithms, and artificial-life > pioneer; professor of computer science and psychology at the U. > of Michigan) > * Frank Drake (distinguished radio-astronomer and head of the SETI > Institute -- Search for Extraterrestrial Intelligence) > * John Koza (inventor of genetic programming, a rapidly expanding > branch of artificial intelligence) > * > > > Symposium organizer and panel moderator: > * Douglas Hofstadter (professor of cognitive science at Indiana; > author of "G del, Escher, Bach", "Fluid Concepts and Creative > Analogies", etc.) > ---------------- > > In 1999, two distinguished computer scientists, Ray Kurzweil and > Hans Moravec, came out independently with serious books that > proclaimed that in the coming century, our own computational > technology, marching to the exponential drum of Moore's Law and more > general laws of bootstrapping, leapfrogging, positive-feedback > progress, will outstrip us intellectually and spiritually, becoming > not only deeply creative but deeply emotive, thus usurping from us > humans our self-appointed position as "the highest product of > evolution". > > These two books (and several others that appeared at about the same > time) are not the works of crackpots; they have been reviewed at > the highest levels of the nation's press, and often very favorably. > But the scenarios they paint are surrealistic, > science-fiction-like, and often shocking. > > According to Kurzweil and Moravec, today's human researchers, > drawing on emerging research areas such as artificial life, > artificial > intelligence, nanotechnology, virtual reality, genetic algorithms, > genetic programming, and optical, DNA, and quantum computing (as > well as other areas that have not yet been dreamt of), are striving, > perhaps unwittingly, to render themselves obsolete -- and in > this strange endeavor, they are being aided and abetted by the very > entities that would replace them (and you and me): superpowerful > computers that are relentlessly becoming tinier and tinier and > faster and faster, month after month after month. > > Where will it all lead? Will we soon pass the spiritual baton to > software minds that will swim in virtual realities of a thousand > sorts that we cannot even begin to imagine? Will uploading and > downloading of full minds onto the Web become a commonplace? Will > thinking take place at silicon speeds, millions of times greater > than carbon speeds? Will our children -- or perhaps our > grandchildren -- be the last generation to experience "the human > condition"? Will immortality take over from mortality? Will > personalities blur and merge and interpenetrate as the need for > biological bodies and brains recedes into the past? What is to come? > > To treat these disorienting themes with the seriousness they deserve > at the dawn of the new millennium, cognitive scientist Douglas > Hofstadter has drawn together a blue-ribbon panel of experts in all > the areas concerned, including the authors of the two books > cited. On Saturday, April 1 (take the date as you will), three main > speakers and five additional panelists will publicly discuss and > debate what the computational and technological future holds for > humanity. The forum will be held from 1 PM till 5:30 PM, and > audience participation will be welcome in the final third of the > program. > > ---------------- > > This event is brought to you with the gracious support of > Stanford's Symbolic Systems Program, > the Center for the Study of Language and Information, > the Computer Science and Philosophy Departments, > the Center for Computer-Assisted Research in the Humanities, > Channel 51, > and the GSB Futurist Club. > > ---------------------------------------------------------------------- > > Message #13409 > From: "john grigg" <> > Subject: Charles, you just gotta believe! > Date: Mon, 20 Mar 2000 13:51:40 PST > > >From: bankston <>> Subject: Re: CryoNet #13399 - > #13401 > >I continue to receive and read many of the CryoNet updates and > would like > >to > add> a few things. I work in an acquired brain injury program in San > Diego > that > works > >every day with over 100 individuals surviving stroke, anoxia, > tramatic > >brain > >injuries, the surgeons byproducts of tumor removals, etc. I can > attest to > >one > >undeniable fact, boys and girls; when areas of the brain are > damaged, > >corelated > >functioning for that particular formation results in flat ass loss. > As in > >forever. > > Charles Platt wrote: > Thanks for this small dose of fact-based reality, which of course > will not > be welcome among those locked in permanent denial. > (end) > > But Charles, nanotech will fix everything!! With our nano and AI we > will > just have to build time machines to find out all the missing > information! > > > your friend, > > John Grigg ;) > > P.S. I am going to start keeping a daily journal so in roughly > seventy > years I can say, "who was this person?" And I would probably say > that > whether I was a healthy but very frail 103 year-old who never took > the 'cold > bath' or a vigorous reanimated amnesiac! > ______________________________________________________ > Get Your Private, Free Email at http://www.hotmail.com > > ---------------------------------------------------------------------- > > Message #13410 > Date: Tue, 21 Mar 2000 00:09:35 -0800 (PST) > From: Doug Skrecky <> > Subject: peg - a non-toxic treatment for colon cancer > > Citations: 1-3 (plus an additional note.) > <1> > Authors > Parnaud G. Tache S. Peiffer G. Corpet DE. > Institution > Securite des Aliments, Institut National de la Recherche > Agronomique, Ecole > Nationale Veterinaire de Toulouse, France. > Title > Polyethylene-glycol suppresses colon > cancer and causes dose-dependent regression of > azoxymethane-induced aberrant crypt foci in rats. > Source > Cancer Research. 59(20):5143-7, 1999 Oct 15. > Abstract > Dietary polyethylene-glycol (PEG) 8000, a > nonfermented polymer laxative, strongly suppresses > azoxymethane-induced > aberrant crypt foci (ACF) in the colon of rats, as shown in a > previous study > (D. E. Corpet et al., Carcinogenesis (Lond.), 20: 915-918, 1999). > In the > present study, we tested the effect of PEG administered during > either > initiation or postinitiation, the dose-response effect of PEG, the > regressive > effect of PEG on established ACF, and the preventive effect of PEG > on colon > cancers in rats. The general design was to initiate > carcinogenesis in F344 rats by a single injection of azoxymethane > (20 mg/kg) > and to randomize the animals 7 days later to AIN-76 diets > containing 5% PEG > or no PEG (control). At termination, ACF and tumors were scored > blindly by a > single observer. The administration of 5% PEG for 32 days to > groups of 10 > female rats in either food or drinking water reduced the number of > ACF by a > factor of 8 (P = 0.0002) and reduced the number of large ACF by a > factor of > 20-30 (P = 0.002). No protection was afforded when PEG was given > only during > the initiation phase. Diets containing 0%, 0.5%, 2%, or 5% PEG fed > for 35 > days to four groups of male rats inhibited ACF in a dose-dependent > manner (P > < 0.0001). The administration of a 5% PEG diet for 41 days, > starting 42 days > after carcinogen injection, led to a 73% decrease in the number of > ACF (P < > 0.0001). Dietary PEG thus caused the regression of established > ACF. > Macroscopic tumors were evaluated by histology in rats that had > been fed a > high-fat diet containing cooked casein to promote tumor growth for > 81 days. > In this accelerated model of carcinogenesis, dietary PEG > suppressed the > occurrence of colon adenomas and carcinomas: the incidence of > tumors > decreased from 70% to 10% (P = 0.005); and the multiplicity > decreased from > 2.1 to 0.1 tumor(s)/rat (P = 0.003). No cancer was detected > in the PEG-fed rats. Taken together, these results suggest that > PEG could be > a potent anticancer agent in the postinitiation phase of > carcinogenesis. Because PEG is a substance that is generally > recognized as > safe (GRAS list, Food and Drug Administration), its > cancer-preventive features could be tested in humans. > > <2> > Authors > Corpet DE. Parnaud G. > Institution > Securite des Aliments, INRA, Ecole Nationale Veterinaire de > Toulouse, France. > > Title > Polyethylene-glycol, a potent suppressor of > azoxymethane-induced colonic aberrant crypt foci in rats. > Source > Carcinogenesis. 20(5):915-8, 1999 May. > Abstract > Bulking fibers and high water intake may decrease colon > carcinogenesis in > rats, and the risk of colorectal cancer in humans. We > speculated that a non-fermented polymer, > polyethylene-glycol (PEG) 8000, which > increases stool moisture, might protect rats against colon > carcinogenesis. > Thirty female F344 rats were given a single injection of > azoxymethane (20 > mg/kg), and 7 days later randomized to AIN76 diets containing PEG > (to provide > 3 g/kg body wt/day), or no PEG (control). Diets were given ad > libitum for 105 > days, then colon carcinogenesis was assessed by the aberrant crypt > foci (ACF) > test. ACF were scored blindly by a single observer. Dietary > feeding of PEG > almost suppressed ACF larger than one crypt, and strikingly > decreased the > total number of ACF per rat. PEG-fed rats had 100 times less large > ACF than > controls (0.8 and 83 respectively, P = 0.00001). PEG-fed rats had > 20 times > less total ACF than control (six and 107 ACF/rat, respectively; P > < 0.0001). > Two treated rats had no detectable ACF. PEG is 10 times more > potent than > other chemopreventive agents in this model. Since PEG is generally > recognized > as safe, its cancer-preventive features could be tested in > humans. > > <3> > Authors > Laboisse CL. Maoret JJ. Triadou N. Augeron C. > Institution > Laboratoire de Biologie et de Physiologie des Cellules Digestives > (U239 > I.N.S.E.R.M.), Faculte X. Bichat, Paris, France. > Title > Restoration by polyethylene glycol of > characteristics of intestinal differentiation in subpopulations of > the human > colonic adenocarcinoma cell line HT29. > Source > Cancer Research. 48(9):2498-504, 1988 May 1. > Abstract > The human colonic cancer cell line HT29 is morphologically > undifferentiated in standard culture conditions. The cells were > incubated for > 30 s in polyethylene glycol (27%, v/v), > then washed, and refed with standard medium. In these conditions > of > treatment, polyethylene glycol was unable > to induce a significant cell multinucleation. Three wk after the > treatment, > circular "flat-foci" developed in the culture, which consisted of > circular > monolayers of polarized cells. These subpopulations were isolated, > then grown > as independent lines (lines 27, 28, 30, and 31) in standard > culture > conditions, and characterized. Two types of differentiated cells > were present > in these lines, namely, enterocytic cells and mucus-secreting > goblet cells. > These characteristics of intestinal differentiation were found to > be stable > during the long-term culture of these lines in standard medium. We > were able > to isolate from line 27 a clonal derivative (C1.27H) exhibiting 2 > lineages of > differentiation, as assessed by electron microscopy, > immunofluorescence, and > immunoblot analysis of cell membranes with anti-sucrase-isomaltase > antibodies, and enzyme activities. Sucrase-isomaltase was present > in two > forms, namely, the high-molecular-weight precursor and the cleaved > subunits. > Finally, the C1.27H cells were found to be significantly less > tumorigenic > than the parental HT29 cells in both in vitro and in vivo > tumorigenicity > tests. This stably differentiated cell clone could represent the > cancer derivative of the normal stem cells of the intestinal > crypt. It is therefore a possible model system for the study of > intestinal > cell differentiation. > > > Additional note by poster: > > High molecular weight peg 8000 appears to be useful in the > treatment > of colon cancer. Considering its very high safety margins relative > to conventional chemotherapy, it deserves first consideration for > further research. (too bad it is not patentable!) > The question I would like to see addressed is whether low > molecular weight peg 200 could be useful in the treatment of other > cancers. Since this would be absorbable from the gastrointestinal > tract, it might be active against a wide variety of tumors. > > ---------------------------------------------------------------------- > > End of CryoNet Digest > ********************* > John W. Perry, Esq., "Failure is impossible!" Mailto: Cellphone E-mail: Mailto: 212-333-3083 office 917-677-8533 fax 917-593-4026 cell 917-875-5322 pager Rate This Message: http://www.cryonet.org/cgi-bin/rate.cgi?msg=13412