X-Message-Number: 31805
Date: Thu, 9 Jul 2009 17:14:08 -0700 (PDT)
From: 
Subject: Rapamycin Extends Lifespan Of Old Mice: 28 To 38 Percent Long...


[Note: A special more bioavailable microencapsulated form of rapamycin was 
used.]

http://www.uthscsa.edu/hscnews/singleformat.asp?newID=3138


Easter Island compound extends lifespan of old mice, scientists report in Nature

Posted: Wednesday, July 08, 2009
Contact: Will Sansom, (210) 567-2579

UT Health Science Center, other centers reach same result: 28%-38% longer life


SAN ANTONIO (July 8, 2009)-The giant monoliths of Easter Island are worn, but 
they have endured for centuries. New research suggests that a compound first 
discovered in the soil of the South Pacific island might help us stand the test 
of time, too.


Today in the journal Nature, The University of Texas Health Science Center at 
San Antonio and two collaborating centers reported that the Easter Island 
compound - called "rapamycin" after the island's Polynesian name, Rapa Nui - 
extended the expected lifespan of middle-aged mice by 28 percent to 38 percent. 
In human terms, this would be greater than the predicted increase in extra years
of life if cancer and heart disease were both cured and prevented.


The rapamycin was given to the mice at an age equivalent to 60 years old in 
humans.


The studies are part of the National Institute on Aging (NIA) Interventions 
Testing Program, which seeks compounds that might help people remain active and 
disease-free throughout their lives. The other two centers involved are the 
University of Michigan at Ann Arbor and Jackson Laboratory in Bar Harbor, Maine.


The Texas study was led by scientists at two institutes at the UT Health Science
Center: the Institute of Biotechnology (IBT) and the Barshop Institute for 
Longevity and Aging Studies.


"I've been in aging research for 35 years and there have been many so-called 
'anti-aging' interventions over those years that were never successful," said 
Arlan G. Richardson, Ph.D., director of the Barshop Institute. "I never thought 
we would find an anti-aging pill for people in my lifetime; however, rapamycin 
shows a great deal of promise to do just that."

Versatile compound


Discovered in the 1970s, rapamycin was first noted for its anti-fungal 
properties and later was used to prevent organ rejection in transplant patients.
It also is used in stents, which are implanted in patients during angioplasty 
to keep coronary arteries open. It is in clinical trials for the treatment of 
cancer.


The new aging experiments found that adding rapamycin to the diet of older mice 
increased their lifespan. The results were the same in Texas, Michigan and 
Maine.


"We believe this is the first convincing evidence that the aging process can be 
slowed and lifespan can be extended by a drug therapy starting at an advanced 
age," said Randy Strong, Ph.D., who directs the NIA-funded Aging Interventions 
Testing Center in San Antonio. He is a professor of pharmacology at the UT 
Health Science Center and a senior research career scientist with the South 
Texas Veterans Health Care System.


The findings have "interesting implications for our understanding of the aging 
process," said Z. Dave Sharp, Ph.D., director of the Institute of Biotechnology 
and professor and chairman of the Health Science Center's Department of 
Molecular Medicine.


"In addition," Dr. Sharp said, "the findings have immediate implications for 
preventive medicine and human health, in that rapamycin is already in clinical 
usage."

Molecular pathway


Aging researchers currently acknowledge only two life-extending interventions in
mammals: calorie restriction and genetic manipulation. Rapamycin appears to 
partially shut down the same molecular pathway as restricting food intake or 
reducing growth factors.


It does so through a cellular protein called mTOR (mammalian target of 
rapamycin), which controls many processes in cell metabolism and responses to 
stress.


A decade ago, Dr. Sharp proposed to his colleagues that mTOR might be involved 
in calorie restriction. "It seemed like an off-the-wall idea at that time," Dr. 
Richardson said.


In 2004, a year after the launch of the NIA Interventions Testing Program, Dr. 
Sharp submitted a proposal that rapamycin be studied for anti-aging effects. The
proposal was approved, and testing centers in San Antonio and elsewhere began 
to include rapamycin in the diets of mice.


The male and female mice were cross-bred from four different strains of mice to 
more closely mimic the genetic diversity and disease susceptibility of the human
population.


Dr. Strong soon recognized a problem: Rapamycin was not stable enough in food or
in the digestive tract to register in the animals' blood level. He worked with 
the Southwest Research Institute in San Antonio to improve the bioavailability 
of the compound through a process called microencapsulation. The reformulated 
drug was stable in the diet fed to the mice and bypassed the stomach to release 
in the intestine, where it could more reliably enter the bloodstream.

Older mice


The original goal was to begin feeding the mice at 4 months of age, but because 
of the delay caused by developing the new formulation, the mice were not started
until they were 20 months old - the equivalent of 60 years of age in humans.

The teams decided to try the rapamycin intervention anyway.


"I did not think that it would work because the mice were too old when the 
treatment was started," Dr. Richardson said. "Most reports indicate that calorie
restriction doesn't work when implemented in old animals. The fact that 
rapamycin increases lifespan in relatively old mice was totally unexpected."


Added Dr. Strong: "This study has clearly identified a potential therapeutic 
target for the development of drugs aimed at preventing age-related diseases and
extending healthy lifespan. If rapamycin, or drugs like rapamycin, works as 
envisioned, the potential reduction in overall health cost for the U.S. and the 
world will be enormous."


Leaders of the other interventions testing centers are Richard Miller, M.D., 
Ph.D., of the University of Michigan and David Harrison, Ph.D., at the Jackson 
Laboratories.


# # #


The University of Texas Health Science Center at San Antonio is the leading 
research institution in South Texas and one of the major health sciences 
universities in the world. With an operating budget of $668 million, the Health 
Science Center is the chief catalyst for the $16.3 billion biosciences and 
health care sector in San Antonio's economy. The Health Science Center has had 
an estimated $36 billion impact on the region since inception and has expanded 
to six campuses in San Antonio, Laredo, Harlingen and Edinburg. More than 25,600
graduates (physicians, dentists, nurses, scientists and other health 
professionals) serve in their fields, including many in Texas. Health Science 
Center faculty are international leaders in cancer, cardiovascular disease, 
diabetes, aging, stroke prevention, kidney disease, orthopaedics, research 
imaging, transplant surgery, psychiatry and clinical neurosciences, pain 
management, genetics, nursing, dentistry and many other fields. For more 
information, visit www.uthscsa.edu.

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