X-Message-Number: 7539
Date: 20 Jan 97 23:09:57 EST
From: Mike Darwin <>
Subject: Cryopatient Temperature Monitoring 

The following is a BioPreservation Technical Briefing 
discussing the rationale for multi-site core temperature 
monitoring in human cryopreservation patients.  This brief 
also discusses the procedures for application of specific 
(and in some cases proprietary) temperature monitoring equipment.

Temperature Monitoring in the Human Cryopreservation 
Patient: Theoretical Considerations and Practical Techniques

by Michael Darwin

Introduction

	The first two pillars upon which cryopatient transport 
rests are cardiopulmonary support and pharmacological 
reduction or elimination of ischemia-reperfusion injury. The 
third pillar of transport is induction of hypothermia. 
Arguably, in many patients, cooling will be the single most 
important injury-mediating modality that can be brought to 
bear. In many patients, application of cardiopulmonary 
support will not be effective or in some cases even possible 
owing to the physical condition of the patient, or the 
logistics of the case. In such instances, rapid induction of 
hypothermia will be the only way possible to slow the pace 
of, and/or reduce the extent of, ischemic injury.

	It is of critical importance to monitor and record the 
patient's core temperatures because this information:

*Constitutes a useful record of that patient's care by 
providing feedback which may allow for real-time 
intervention to improve care.

*Will likely aide future attempts at recovery by 
contributing to the documentation of the patient's overall 
condition.

*Provides a medicolegal record of a critical intervention 
(induction of hypothermia) which is central to providing 
good care to the patient.

*Allows for evaluation of the efficacy of the techniques 
applied on a given patient and contributes to the creation 
of a statistically meaningful database with which to 
evaluate the efficacy of given interventions.

*Facilitates evaluation of the effectiveness of 
cardiopulmonary support and the duration of post arrest 
cerebral perfusion.

*Allows for improved differential diagnosis in the 
interpretation of physical signs which may be present during 
the course of the patient's care such as rigor, secondary 
flaccidity, clotting, etc.


Calibration of Temperature Probes

	Thermocouple temperature probes (the kind used 
exclusively in cryopreservation operations) consist of two 
different kinds of metals which are welded together at a 
junction. This bimetallic junction generates a minute 
electrical current which varies with the temperature of the 
junction. Thus, by measuring the current at the weld-point 
or junction, it is possible to measure the temperature of 
the junction. This is the principle on which thermocouple 
thermometry operates. The kind of thermocouple used in human 
cryopreservation operations is the copper-constantan 
thermocouple (Cu++/C) which has a useful range of 
temperature measurement from well above body temperature, to 
a little below liquid nitrogen temperature, with theoretical 
accuracy to a fraction of a degree Celsius.

	Like most other measuring equipment, thermocouple probes 
must be calibrated prior to use, or, if pre-calibrated, 
their accuracy must be determined before they are used. For 
transport procedures a one-point calibration or accuracy 
check on the probes using a slurry of crushed ice and water 
is sufficient.

	To carry out a one-point calibration with maximum 
accuracy it is best to use an insulated container (even a 
foam coffee cup will suffice) which is filled with crushed 
ice and sufficient water to convert the ice into a slush. 
The probe tip should then be held in the center of this bath 
of ice-slush until the temperature stabilizes at 0 C +/- 
0.5 C.  If a probe fails to pass this test, another probe 
which does read appropriately should be substituted.  

	Alternatively, if the thermocouple meter has provisions 
for doing so, the meter should be re-calibrated by resetting 
the measured temperature to 0 C (see individual meter 
instructions for calibration procedures). If re-calibration 
or probe replacement is not possible, and the probe must be 
used, it is essential to note the degree of offset (i.e., 
inaccuracy) from 0 C in the patient temperature descent log, 
and to tag the probe immediately and indelibly so that a 
corrected cooling curve can be constructed using the probe 
after the conclusion of transport and removal of the probe 
from the patient. An incorrectly reading probe should not be 
left in the patient during subsequent cryoprotective 
perfusion or subzero cooling operations.

Note: Specific instructions for calibration and use of the 
particular model of thermocouple meter now in use by BPI and 
its client cryonics organizations is provided in specific 
detail below. When using other equipment it is important to 
follow the manufacturer's instructions carefully. For this 
reason it is recommended that a copy of all instructional 
materials/manuals provided by the manufacturer be kept with 
the equipment to be used in the field: if possible by 
attaching the manual or a reduced-sized photocopy of it to 
the associated piece of equipment.

Rationale for Multi-Site Core Temperature Monitoring

	The protocol specified here calls for monitoring the 
patient's core temperature at multiple sites including both 
tympanic membranes, nasopharyngeal (using a the left or 
right nare for probe access), deep-pharynx or esophagus, 
and, if possible and appropriate, the rectum. While this may 
seem burdensome and of questionable value at a time when 
almost all available resources will likely be focused on 
carrying out cooling and providing cardiopulmonary support, 
there are very good reasons for this approach.

Figure 8-1: Esophageal and tympanic temperature cooling 
curves for a dog subjected to five hours of asanguineous 
extracorporeal perfusion with subsequent rewarming. This 
figure shows very tight (to with 0.2 degrees C)correlation 
of typmanic and esophageal temperature. 


	In patients experiencing pre-arrest shock and 
hypotension even distribution of blood flow is disrupted. 
Cardiac arrest and any accompanying ischemic interval only 
serve to exacerbate this problem. In the case of the rectum, 
retained or impacted fecal material may encase the 
temperature probe upon insertion and render its readings 
inaccurate. Further, shock-induced compromise of blood flow 
to the bowel and rectal vascular plexus may selectively slow 
cooling at this site yielding data which is not 
representative of the rest of the body and in particular the 
brain,  and thus is unreliable.

	The obverse of this may also be true in that the rate of 
cooling observed rectally during femoral-femoral 
cardiopulmonary bypass and extracorporeal cooling is often 
far more rapid than that observed in the brain, or when 
measured esophageally or pharyngeally.

	Another very significant problem with single-site 
monitoring of core temperature is that if a probe is 
malpositioned, becomes inaccurate, or develops an 
intermittent failure mode, there are no other probes to act 
as back-ups or to allow cross-checking of data. Further, new 
cooling modalities such as liquid ventilation, gastric, 
colonic, and peritoneal iced-fluid lavage may render 
esophageal and rectal temperature measurements meaningless.

	Finally, the use of bilateral tympanic temperature 
monitoring is of critical importance for several reasons 
which deserve both discussion and illustration with actual 
case data. In a hemodynamically intact animal or human, 
tympanic and esophageal temperatures correlate very well as 
is shown in Figure 8-1. Esophageal temperature can 
reasonably be taken to be indicative of intrathoracic and 
large-vessel blood temperatures (the esophagus is contained 
within the mediastinum in close proximity to the aorta and 
the superior and inferior vena cava).  

	The brain core temperature and the brain cortical 
(surface) temperature (the latter only in the case of a 
patient with significant cerebral perfusion) will be most 
accurately reflected by the tympanic temperature since the 
blood supply for both the brain and tympanic membranes, and 
the associated thermal-dilutional effects will be more 
representative than those of the hemorrhoidal venous plexus 
or the esophagus. Clinical and experimental data indicate a 
closer correlation of tympanic temperature to brain and 
central venous temperature than is seen for rectal 
temperature 1,2,3.  Tympanic temperature may vary 
significantly from that cerebral cortical temperature in the 
clinical setting4, and may vary more so in cryopatients, 
however, it still likely to be far more accurate in 
representing overall brain temperature than is either 
esophageal or rectal thermometry. (We have demonstrated that 
this is the case in-house in canine models of both surface 
and extracorporeally supported cooling in ischemia).

Figure 8-2: Cooling curves for patient C-2150 demonstrating 
failure and resumption of cerebral perfusion as illustrated 
by the plateau in tympanic temperatures observed during the 
latter part of HI-ACD-CPR and continuing until the start of 
extracorporeal perfusion.

	It is a well established fact in the literature that 
cerebral blood flow is not maintained in conventional CPR 
for more than 10 to 15 minutes 5,6. Blood flow to visceral 
organs is maintained for considerably longer periods of 
time. Failure of cerebral perfusion can be documented by 
frequent sampling of tympanic, esophageal, and/or hepatic 
temperatures and plotting of these temperatures on a common 
graph. Failure of cerebral perfusion is demonstrated by the 
development of a discontinuity marked by failure of the 
tympanic temperatures to track the other core temperatures 
during non-extracorporeal supported cooling, and a 
resumption of comparable rates of cooling once adequate mean 
arterial pressure (MAP) and flow are restored by 
extracorporeal means. The caveat being that such restoration 
of MAP and flow occur before supervening processes render 
re-establishment of cerebral perfusion impossible (clotting, 
cerebral edema, etc.). This phenomenon is shown quite well 
in the temperature data collected from patient C-2150 shown 
in Figure 8-2.

End of Part I

BioPreservation, Inc
10743 Civic Center Drive
Rancho Cucamonga, CA 91730
(909)97-3883


Rate This Message: http://www.cryonet.org/cgi-bin/rate.cgi?msg=7539